MSG: The Chemical and Biological Reactions

Mono atomic fig 11 glutam have (monosodium glutamate) is a flavorless chemical added to a variety of foods. time in that respect is no 100 percent conclusive study that represents monosodium glutamate as harmful, it is widely linked to seizures, chest pain, and other adverse slope effects in adults and children. In this paper, I will show how the interpersonal chemistry of monosodium glutamate causes a negative biological reaction.N. Peica, C. Lehene, N. Leopold, S. Schlucker, and W. Kiefer (2007) wrote, monosodium glutamate or E621, the sodium salt of glutamic acid, is probably the most common food additive utilize in m any packaged and prep bed foods (p. 604). Because of this, it is easy for monosodium glutamate to hide. This is peculiarly problematic for people who have a sensitivity to MSG. Reporter Sandra Blakeslee (1990) wrote, several(prenominal) months ago Jack Samuels, a Chicago businessman, carefully read the differentiate on a box of breakfast cereal and decided i t was preventative to eat.An hour later, however, Mr. Samuels collapsed in his physicians office after a treadmill test. As I started to lose consciousness, I knew it was an MSG attack, he said (p. C3). The article went on to report that the FDA allows several different name for MSG sometimes the pseudonyms make it difficult for consumers to realize they are ingesting MSG.In its monohydrate form, the two carboxy groups of MSG twist to an equal scarcely opposite purpose with respect to the carbon chain (Peica et al. 2006, p. 606). In its anhydrous form, Anhydrous MSG is arched and two hydrogen bond interactions are possible (Peica et al. 2006, p. 607). both way, the result is the formation of white crystals. The crystals dont have any flavor however, its chemical pay back allows it to enhance the flavor of other foods. MSG concentrations can be as low as 10-5, or overmuch higher.Michelle Meadows (2003) expressed that the 1995 FASEB report confirmed that MSG is safe if consumed moderately. She added that there is a group of people who is sensitive to MSG if more than 3 grams are consumed at a meal (a typical serving is 0.5 grams). She wrote that some solemn and poorly controlled asthmatics alike have trouble with MSG consumption. For them, the lack of labeling is problematic because labels only have to reflect foods that have MSG added to them, not foods that have got it naturally, like tomatoes, cheeses, soy sauce, and some meats.Chris Kilham (2006) pointed out that a vast number of foods contain MSG, although there are plenty of studies that sustain its harmful effects. He wrote, Studies have shown that mice fed large amounts of MSG suffer destruction of top dog cells Several years ago, bollocks up food manufacturers stopped using MSG under intense public pressure. However, MSG remains in touristed use (p. 25). He added that the risk of using MSG is unnecessary, since it has absolutely no nutritional value.Angelica Soares, Joao Paulo Ferreira S choffen, Elsa Maria De Gouveia, and Maria Raquel Marcal Natali (2006) also looked at the worrying effect MSG had on rats. They studied infant rats and launch that the embayment rats that ingested MSG showed high levels of NADH-diaphorase. The NADH caused philia damage, as well as caused the baby rats to retain more fat tissue that the ones not given MSG. The death of the study was that not only did MSG contribute to obesity it also caused a deterioration in the intestinal lining.M. Hermanussen, A.P. Garcia, M. Sunder, M. Voigt, V. Salazar, and J.A.F. Tresguerres (2006) agreed with Soares et al (2006), but added some interesting findings of their own. They looked at glutamic acid (GLU), since it is a major fragment in glutamine. The study started with administering MSG to pregnant rats, then to the offspring of the rats. Obviously, there was a control group not receiving MSG, except what was normally found in the regular feed.The conclusion was that male rats showed a lot more damage from the MSG than female rats did. Obesity was the obvious result, but, in part, because the MSG-fed rats ate more. This indicated that GLU significantly increased appetite. The group noted, too, that the MSG-fed rats retained much more adipose tissue that the rats that werent fed MSG. As well, it was noted that the same nerve damage found in the study by Soares et al. (2006) was consistent with the findings by Hermanussen et al. (2006). Hermanussen et al. clearly discovered impaired glucose tolerance and insulin resistance in their rats, as a result of GH burst frequency (p. 30).Despite statements that there is no definite evidence that MSG causes negative reactions, very recent studies prove the contrary. Since MSG does not add any nutritional value to food, it capacity be wise to avoid it, especially in large quantities. The studies show that MSG is linked to nerve damage, obesity, voracity, and diabetes. We also know that chemical make-up of the product is more likely t o affect men and asthmatics meaning that a significant number of Americans may suffer adverse reaction s from MSG, and some of them dont even know why.ReferencesBlakeslee, S. (1990, environ 6). With MSG sensitivity still at issue in studies, label rules tighten. modern York Times, p. C3. Retrieved Thursday, March 29, 2007 from the ProQuest database.Hermanussen, M., Garcia, A.P., Sunder, M., Voigt, M., Salazar, V., & Tresguerres, J.A.F.(2006). Obesity, voracity, and short stature The impact of glutamate on the regulation of appetite. European Journal of Clinical Nutrition, 60, 25-31. Retrieved Thursday, March 29, 2007, from the Academic Search Premier database.Kilham, C. (2006, March/April). The dangers of chemical food additives. Share Guide, p. 25.Meadows, M. (2003, January/February). A common flavor enhancer. FDA Consumer, p. 35.Pieca, N., Lehene, C., Leopold, N., Schlucker, S., & Kiefer, W. (2006). Monosodium glutamatein its anhydrous and monohydrate form Differentiation by Ram an spectroscopies and density functional calculations. Spectrochimica Acta lead off A, 604-615. Retrieved Thursday, March 29, 2007, from the ScienceDirect database.Soares, A., Schoffen, J.P.F., De Gouveia, E.M., & Natali, M.R.M. (2006). Effects of the neonataltreatment with monosodium glutamate on myenteric neurons and the intestinal smother in the ileum of rats. Journal of Gastroenterology, 41, 674-680. Retrieved Thursday, March 29, 2007, from the Academic Search Premier database.